Sanjabi S, Oh SA, Li MO. Regulation of the Immune Response by TGF-β: From Conception to AutoImmunity and Infection. Cold Spring Harb Perspect Biol. 2017 Jan 17 pii: a022236. doi: 10.1101/cshperspect.a022236
Khan S, Woodruff EM, Trapecar M, Fontaine KA, Ezaki A, Borbet TC, Ott M, Sanjabi S. Dampened antiviral immunity to intravaginal exposure to RNA viral pathogens allows enhanced viral replication. The Journal of Experimental Medicine. 2016 Nov 16;10.1084
Ishigame H, Mosaheb MM, Sanjabi S*, Flavell RA*. Truncated Form of TGF-βRII, But Not Its Absence, Induces Memory CD8+ T Cell Expansion and Lymphoproliferative Disorder in Mice. Journal of Immunology. 2013 Jun 15;190(12):6340-50. *Co-corresponding
Ishigame H, Zenewicz LA, Sanjabi S, Licona-Limón P, Nakayama M, Leonard WJ, Flavell RA. Excessive Th1 responses due to the absence of TGF-β signaling cause autoimmune diabetes and dysregulated Treg cell homeostasis. Proceedings of the National Academy of Sciences U. S. A. 2013 Apr 23;110(17):6961-6.
Flavell RA, Sanjabi S, Wrzesinski S, and Licona-Limon P. Polarization of immune cells in the tumour environment by TGF-β. Nature Reviews Immunology. 2010, Aug;10(8):554-67.
Sanjabi S, and Flavell RA. Interaction between TGF-β and the common gamma chain cytokines during an inflammatory immune response. Cell Cycle. 2010, 9(3): 428-30.
Sanjabi S, and Flavell RA. Overcoming the hurdles in using mouse genetic models that block TGF-β signaling.Journal of Immunological Methods. 2010, 353(1-2): 111-114.
Sanjabi S, Mosaheb MM, and Flavell RA. Opposing effects of TGF-β and IL-15 cytokines control the number of short-lived effector CD8+ T cells. Immunity. 2009, 31(1): 131-44.
Matza D, Badou A, Jha M, Willinger T, Antov A, Sanjabi S, Kobayashi K, Marchesi V, and Flavell, RA. Requirement for AHNAK1-mediated calcium signaling during T lymphocyte cytolysis. Proceedings of the National Academy of Science U. S. A. 2009, 106(24): 9785-90.
Sanjabi S, Zenewicz LA, Kamanaka M, and Flavell RA. Anti-inflammatory and pro-inflammatory roles of TGF-β, IL-10, and IL-22 in immunity and autoimmunity. Current Opinion in Pharmacology. 2009, 9(4):447-53.
Wang J, Wang X, Hussain S, Zheng Y, Sanjabi S, Ouaaz F, and Beg AA. Distinct roles of different NF-κB subunits in regulating inflammatory and T cell stimulatory gene expression in dendritic cells. Journal of Immunology. 2007, 178(11): 6777-88.
Li MO, Sanjabi S, and Flavell RA. Transforming Growth Factor-β Controls Development, Homeostasis, and Tolerance of T Cells by Regulatory T Cell-Dependent and -Independent Mechanisms. Immunity. 2006, 25(3): 455-71.
Kriegel MA, Li MO, Sanjabi S, Wan YY, and Flavell RA. Transforming growth factor-β: recent advances on its role in immune tolerance. Current Rheumatology Reports. 2006, 8(2): 138-44.
Li MO, Wan YY, Sanjabi S, Robertson AK, and Flavell RA. Transforming growth factor-β regulation of immune responses. Annual Reviews of Immunology. 2006, 24: 99-146.
Dong C, Sanjabi S, and Eynon EE. Gene regulation and function: its rocking science. Immunity. 2005, 23(6):557-60.
Sanjabi S, Williams KJ, Saccani S, Zhou L, Hoffmann A, Ghosh G, Gerondakis S, Natoli G, and Smale ST. A c-Rel subdomain responsible for enhanced DNA-binding affinity and selective gene activation. Genes & Development. 2005, 15;19(18): 2138-51.
Weinmann AS, Mitchell DM, Sanjabi S, Bradley MN, Hoffmann A, Liou HC, and Smale ST. Nucleosome remodeling at the IL-12 p40 promoter is a TLR-dependent, Rel-independent event. Nature Immunology. 2001, 2(1): 51-7.
Sanjabi S, Hoffmann A, Liou HC, Baltimore D, and Smale ST. Selective requirement for c-Rel during IL-12 P40 gene induction in macrophages. Proceedings of the National Academy of Science U. S. A. 2000, 7;97(23): 12705-10.